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Re: CWD in Texas [Re: jmh004] #5834898 07/16/15 09:36 PM
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Originally Posted By: jmh004
Ok, so 8 confirmed cases against the millions of deer in Texas. Sounds unbelievably rare to me.



remember what I said, the key word there was _documented_ cases of cwd in Texas to date, is at 8 cases. CWD has been waltzing across the border into Texas around the WSMR for over a decade, and the testing in captives and the wild is not sufficient, in my opinion, and please don't give me the bs that these farmers are testing all their captive deer. they do NOT test the escapees. ...



Tuesday, July 14, 2015

TWO Escaped Captive Deer on the loose in Eau Claire County Wisconsin CWD postive farm Yellow ear tag

http://chronic-wasting-disease.blogspot.com/2015/07/two-escaped-captive-deer-on-loose-in.html


kind regards, terry

Re: CWD in Texas [Re: Frio County Hunts] #5834900 07/16/15 09:38 PM
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Indeed they have potential....so does the earth to destroy itself from within..

I'm sorry about what happened to your mother and my condolences. But you are running way off the track comparing beef that were fed beef that produced an anomaly that killed a very few people when looking at population numbers.

Please stop it. And let's get the train back on the track.


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Re: CWD in Texas [Re: jmh004] #5834909 07/16/15 09:41 PM
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Originally Posted By: jmh004
Sound like they had a compromise in Austin. Tpwd will not slaughter the entire heard. They will kill and test the 29 deer closest to the positive deer. If they get another positive test, they move on to the next closest deer, and so on. A vast majority of breeders can move deer again. The only ranches that cant yet are the ones that bought and sold deer to and from that ranch in Medina.

Apparently the commissioners who made the decision were swayed by a vet who gave testimony about a live test that is 97% accurate. The vet said Texas can lead the nation in CWD research by using science, not slaughtering hundreds of animals.

So it sounds like the breeders won this one. Hopefully Texas does lead the nation in research of this disease. God knows it has the opportunity to do so with all the deer on farms in this state.



if that is true, Texas just opened up the door for cwd tse prion disease to be trucked/spread to every state that has anything to do with Texas. but that's not unusual, they did this with BSE tse prion as well. nothing knew, just political and corporate junk science at it's best, and Texas is full of that. it's what happens when you let corporations run the government. good luck with that. ...


kind regards, terry


Last edited by flounder; 07/16/15 09:42 PM.
Re: CWD in Texas [Re: Pitchfork Predator] #5834919 07/16/15 09:46 PM
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Originally Posted By: Pitchfork Predator
Indeed they have potential....so does the earth to destroy itself from within..

I'm sorry about what happened to your mother and my condolences. But you are running way off the track comparing beef that were fed beef that produced an anomaly that killed a very few people when looking at population numbers.

Please stop it. And let's get the train back on the track.



I believe you are wrong in your assumptions sir. please see ;



O.05: Transmission of prions to primates after extended silent incubation periods: Implications for BSE and scrapie risk assessment in human populations

Emmanuel Comoy, Jacqueline Mikol, Valerie Durand, Sophie Luccantoni, Evelyne Correia, Nathalie Lescoutra, Capucine Dehen, and Jean-Philippe Deslys Atomic Energy Commission; Fontenay-aux-Roses, France

Prion diseases (PD) are the unique neurodegenerative proteinopathies reputed to be transmissible under field conditions since decades. The transmission of Bovine Spongiform Encephalopathy (BSE) to humans evidenced that an animal PD might be zoonotic under appropriate conditions. Contrarily, in the absence of obvious (epidemiological or experimental) elements supporting a transmission or genetic predispositions, PD, like the other proteinopathies, are reputed to occur spontaneously (atpical animal prion strains, sporadic CJD summing 80% of human prion cases). Non-human primate models provided the first evidences supporting the transmissibiity of human prion strains and the zoonotic potential of BSE. Among them, cynomolgus macaques brought major information for BSE risk assessment for human health (Chen, 2014), according to their phylogenetic proximity to humans and extended lifetime. We used this model to assess the zoonotic potential of other animal PD from bovine, ovine and cervid origins even after very long silent incubation periods. We recently observed the direct transmission of a natural classical scrapie isolate to macaque after a 10-year silent incubation period, with features similar to some reported for human cases of sporadic CJD, albeit requiring fourfold longe incubation than BSE. Scrapie, as recently evoked in humanized mice (Cassard, 2014), is the third potentially zoonotic PD (with BSE and L-type BSE), ***thus questioning the origin of human sporadic cases. We will present an updated panorama of our different transmission studies and discuss the implications of such extended incubation periods on risk assessment of animal PD for human health.

===============

***thus questioning the origin of human sporadic cases...TSS

===============


ALSO, PLEASE SEE ;

31 Jan 2015 at 20:14 GMT

*** Ruminant feed ban for cervids in the United States? ***

31 Jan 2015 at 20:14 GMT

http://www.plosone.org/annotation/listThread.action?root=85351

http://chronic-wasting-disease.blogspot.com/

Saturday, May 30, 2015

PRION 2015 ORAL AND POSTER CONGRESSIONAL ABSTRACTS

https://prion2015.files.wordpress.com/2015/05/prion2015abstracts.pdf

http://transmissiblespongiformencephalopathy.blogspot.com/2015/05/prion-2015-oral-and-poster.html


kind regards, terry

Re: CWD in Texas [Re: Frio County Hunts] #5834924 07/16/15 09:48 PM
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Wow, bitter much? Sounds like Texas just looked and what other states have done in the past and saw that none of that actually worked. Kudos to the state for not listening to people who are paranoid and post links from blogs like they're Bill Nye the Science Guy. If people don't like how the state is run, there's always the option to move to California or Russia.

Re: CWD in Texas [Re: jmh004] #5834930 07/16/15 09:50 PM
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Originally Posted By: jmh004
Sound like they had a compromise in Austin. Tpwd will not slaughter the entire heard. They will kill and test the 29 deer closest to the positive deer. If they get another positive test, they move on to the next closest deer, and so on. A vast majority of breeders can move deer again. The only ranches that cant yet are the ones that bought and sold deer to and from that ranch in Medina.

Apparently the commissioners who made the decision were swayed by a vet who gave testimony about a live test that is 97% accurate. The vet said Texas can lead the nation in CWD research by using science, not slaughtering hundreds of animals.

So it sounds like the breeders won this one. Hopefully Texas does lead the nation in research of this disease. God knows it has the opportunity to do so with all the deer on farms in this state.


cheers texas


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Re: CWD in Texas [Re: Frio County Hunts] #5834939 07/16/15 09:56 PM
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Terry,

I suggest you evaluate what your doing. It's not effective or convincing.

It's become irritating.

kind regards,

Marc


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Re: CWD in Texas [Re: jmh004] #5834940 07/16/15 09:57 PM
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Originally Posted By: jmh004
Wow, bitter much? Sounds like Texas just looked and what other states have done in the past and saw that none of that actually worked. Kudos to the state for not listening to people who are paranoid and post links from blogs like they're Bill Nye the Science Guy. If people don't like how the state is run, there's always the option to move to California or Russia.


yep, that's working out real well for Wisconsin. ...


Wednesday, March 04, 2015

Disease sampling results provide current snapshot of CWD in Wisconsin finding 324 positive detections statewide in 2014

http://chronic-wasting-disease.blogspot.com/2015/03/disease-sampling-results-provide.html


Wisconsin : 436 Deer Have Escaped From Farms to Wild

Date: March 18, 2003 Source: Milwaukee Journal Sentinel

Contacts: LEE BERGQUIST lbergquist@journalsentinel.com

State finds violations, lax record keeping at many sites, report says A state inspection of private deer farms, prompted by the discovery of chronic wasting disease, found that 436 white-tailed deer escaped into the wild, officials said Tuesday

The Department of Natural Resources found that captive deer have escaped from one-third of the state's 550 deer farms over the lifetime of the operations. The agency also uncovered hundreds of violations and has sought a total of 60 citations or charges against deer farm operators.

Hundreds of deer escape

The DNR found a total of 671 deer that escaped farms - 436 of which were never found - because of storm-damaged fences, gates being left open or the animals jumping over or through fences.

In one example in Kewaunee County, a deer farmer's fence was knocked down in a summer storm. Ten deer escaped, and the farmer told the DNR he had no intention of trying to reclaim them. The DNR found five of the deer, killed them and cited the farmer for violation of a regulation related to fencing.

Another deer farmer near Mishicot, in Manitowoc County, released all nine of his whitetails last summer after he believed the discovery of chronic wasting disease was going to drive down the market for captive deer.

The DNR found 24 instances of unlicensed deer farms and issued 19 citations.

Game Farms Inspected

A summary of the findings of the Department of Natural Resources' inspection of 550 private white-tailed deer farms in the state: The deer farms contained at least 16,070 deer, but the DNR believes there are more deer in captivity than that because large deer farms are unable to accurately count their deer. 671 deer had escaped from game farms, including 436 that were never found.

24 farmers were unlicensed. One had been operating illegally since 1999 after he was denied a license because his deer fence did not meet minimum specifications.

Records maintained by operators ranged from "meticulous documentation to relying on memory." At least 227 farms conducted various portions of their deer farm business with cash. Over the last three years, 1,222 deer died on farms for various reasons. Disease testing was not performed nor required on the majority of deer. Farmers reported doing business with people in 22 other states and one Canadian province. ..

http://www.cwd-info.org/index.php/fuseaction/news.detail/ID/4eb67da18ca2c69fce5b5f2eaad058e8

How’s that Texas Deer Czar expert Dr. Dough that Governor Scott Walker hired, how is that working for you now in Wisconsin $$$

Escaped Captive Deer on the loose in Eau Claire County

By Central Office July 14, 2015 Contact(s): Bill Hogseth, DNR Wildlife Biologist, Eau Claire & Chippewa counties, 715-839-3771 Harvey Halvorsen, DNR Area Wildlife Supervisor, 715-684-2914 Ext. 113 Tami Ryan, DNR Wildlife Health Section Chief, 608-266-3143

EAU CLAIRE, Wis. -- The state Department of Natural Resources is requesting the help of residents of Fairchild and Augusta and the surrounding areas to be on the lookout for two escaped ear-tagged captive white-tailed deer from a local captive deer facility.

On June 24 the Department of Agriculture, Trade, & Consumer Protection [PDF] (exit DNR ) announced a captive white-tailed deer from a breeding farm in Eau Claire County has tested positive for chronic wasting disease.

In early May, the farm owner reported that multiple captive bucks escaped the facility when a tree fell on the fence causing a breach. Most of the escaped bucks were recovered with two still remaining out on the landscape.

"We need landowners and the public to be on the lookout for any deer that appear to have an ear tag. These captive escapes are a potential health risk to the local wild deer herd and should be removed from the landscape," said DNR wildlife biologist Bill Hogseth.

Ear tag Yellow plastic ear-tags are likely to be on these two bucks. WDNR Photo

Landowners are asked to check trail camera images for any ear-tagged deer. The DNR would like to be notified if you record any images of ear-tagged deer or if you observe any. While yellow plastic ear-tags like the one pictured are most common and likely to be on these two bucks, please report any deer tagged with any size, shape or color of ear-tag.

If it is after hours, or a biologist isn't available please contact the department's hotline at 1-800-847-9367. The information will be forwarded to the local conservation warden.

Anyone interested in learning more about CWD in Wisconsin can search the DNR website, dnr.wi.gov, for keyword "CWD").

Last Revised: Tuesday, July 14, 2015

http://dnr.wi.gov/news/releases/article/?id=3650

http://datcp.wi.gov/news/



CHRONIC WASTING DISEASE CWD WISCONSIN Almond Deer (Buckhorn Flats) FarmUpdate DECEMBER 2011The CWD infection rate was nearly 80%, the highest ever in a North American captive herd. RECOMMENDATION: That the Board approve the purchase of 80acres of land for $465,000 for the Statewide Wildlife Habitat Program inPortage County and approve the restrictions on public use of the site.SUMMARY:

http://dnr.wi.gov/about/nrb/2011/december/12-11-2b2.pdf




kind regards, terry



Last edited by flounder; 07/16/15 09:58 PM. Reason: added link to dnr buckhorn flats
Re: CWD in Texas [Re: Frio County Hunts] #5834948 07/16/15 10:00 PM
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Dude, please stop posting the same posts repeatedly. We got it, really.

Re: CWD in Texas [Re: Frio County Hunts] #5835354 07/17/15 01:54 AM
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Here is a long read that I received in an email from a testimony that was given on Monday in Austin. He is just stating facts as he sees them.
Quote:
Testimony before Texas House of Representatives Committee on Culture Recreation & Tourism
Monday July 13, 1 pm
Mister Chairman Guillen
Madam Vice Chair Dukes
Esteemed members of the Committee:
Representatives Frullo,Larson,Marquez,Murray, and Smith. QALadies and gentlemen:
Thank you for inviting me to appear before this august body today.
My name is Greg Stewart.
I live in the world of translational medicine and infectious disease. I come from a long line of
physicians and my children continue that tradition. Both currently are in medical school. One is a
senior. The other is in a clinician scientist training (MD/PhD combined) program.
First you have asked me to qualify myself for this role.
I submitted for your review a short synopsis of a much longer curriculum vitae. This you can
peruse at your leisure. My legal residence is in rural Oconee County, Georgia. I work in Texas
between two and three months per year. So howdy!
I hold a Masters of Science in livestock and poultry. I hold a PhD in medical microbiology which
was heavily loaded with pathology, diagnostics, laboratory techniques, and epidemiology.
I hold a Doctor of Veterinary Medicine degree and am licensed to practice in this state and
others. I'm a former faculty member of University of Georgia's College of Veterinary Medicine.
My home department there is called the Department of Population Medicine.I have worked as a
clinician in a very specialized training program for graduate veterinarians and was an integral
part of its diagnostic laboratory system.
I have been for 38 years a servant to production animal medicine. I have been blessed to be able
to design, implement, and supervise "specific pathogen free" programs on multiple species on all
seven continents. This involved surveillance, diagnostics, pathogenesis, biosecurity, genetic
susceptibility, understanding of multiplicity of infection, and modes of transmission.
My family are shepherds. We are primary breeders of fine single purpose meat sheep.
In my veterinary practice we deal daily with decisions, questions, comments, and government
compliance issues relating to a Transmissible Spongiform Encephalopathy(TSE) agent called
scrapie(sheep and goats) and another TSE called Chronic Wasting Disease of Cervids (CWD).
These are also called prion diseases. We routinely test all of our own breeding stock for genetic
resistance to scrapie and the older ewes are tested post mortem for scrapie. There is an USDA
surveillance program governing this agent just as there is one governing Chronic Wasting
Disease. CWD is the subject of this hearing.
I help teach a class with the College of Veterinary Medicine UGA each year in which we train
graduating veterinarians about TSEs and how to monitor flocks and herds using rectal biopsy,
genetic susceptibility testing, and immunohistochemical (IHC) techniques used on collected post
mortem tissues.
For the past seventeen years I have served the Texas small ruminant industry (sheep, goats, and
deer) in various capacities relating to wellness, nutrition, herd health, and reproduction. The
cervid industry is served by our practice in ten or more states each year. We have contributed
considerable resources to public education about CWD. Our website,www.southernvet.co,
contains a reasonably current lecture on the subject. We have cooperated with several television
producers to educate the general public about CWD. We stay current with most newly published
material on the topic. I am knowledgeable of the USDA-APHIS CWD program. I have,for four
years, served as a technical resource for members of Georgia's House of Representatives and
Senate in their evaluation of the risks and benefits of deer farming as they contemplate it.
Secondly you have asked about recommendations about how to eradicate or control CWD.
Let us confine our remarks to management and control because I know of no location or agency
in North America that would lay claim to eradication. In fact, those who have tried are now
changing their strategy towards management and control.
In any "management and control" program, stakeholders must lay down some operational
paradigms and get agreement upon them before that program can be successful.
1. There must be agreement amongst ALL that the work and the plan must be objective and
science based.
2. ALL must agree that "time is of the essence" in dealing with quarantines, release of
quarantines, notifications, and laboratory testing.
3. ALL should agree that if better knowledge and understanding of the disease agent can be
gleaned in the exercise , that we are obligated to seek that knowledge so long as it doesn't
co-opt the program, endanger other at risk animals, or the public health.
4. There must be agreement that bureaucratic turf and competing agency agenda have NO
role in disease control strategies that have as their highest priority objectivity and good
science.
5. The public is a stakeholder and public education about the risks and benefits of the
control program is crucial. There should be ONE assigned, knowledgeable, qualified,
unified voice that is the source of that education and other releases to the public from the
state government.
6. ALL should agree that “the fix" of the problem should NOT be more deadly or
devastating than the disease.
7. ALL must agree to respect the dignity of the animals involved and respect the dignity of
the ranchers involved who have cared for these animals throughout their lives. We must
respect the dignity of the human animal bond. If we do not, we will lessen ourselves in
eyes of the public and our creator. There is precedent for these concerns that arises from
the mass slaughter of animals in the Foot and Mouth Disease outbreak in the UK. If
animals are sacrificed and they will be, we must seek ALL possible knowledge that
furthers our understanding of the disease otherwise that animal's life has been assigned
lesser value than it should have been. All sacrificed animals should be genotyped at
codons 95 & 96 and that information catalogued with its unique DNA information about
parentage.Each should undergo state of the art antemortem testing prior to be being
sacrificed. Retropharyngeal lymph nodes,palatine tonsil, and obex should be harvested
post mortem. As these ranches are visited, biosecurity should play a leading role in
countering transmission from one ranch to another and out to the general environs.
People and equipment are highly efficient fomites and vectors.
The refereed published evidence about CWD contains a plethora of information about the
disease called CWD. Simply put prion diseases are actually proteins that become misfolded.
They create problems when normal conformation changes because structure in protein is part of
function.Vast expenditures have been let in a quest for understanding. My mission here is not to
regurgitate these reviews but to try to put a few things in perspective for your deliberations.
We must seek actual risk and not theoretical risk.
Routes of infection. There are major differences in natural routes of exposure to CWD, such as
ingestion, and unnatural or artificial routes of infection, such as injection of infected material into
the brain of a target species. Our priorities should be on natural routes of exposure.
Exposure does not mean infection.
The incubation period of CWD is quite long compared to viral or bacterial or fungal etiologies.
A positive diagnosis made by IHC staining of tissues occurs long before any clinical signs of the
disease are evident.
We don't completely understand the agent or all the modes of transmission. We are learning
more each year. The agent can be spread by live animal to animal contact, through contaminated
soil, urine, feces, saliva, and recently it appears that plants can uptake the prion from either
contaminated soil or by contaminated sprays or washings of leaves and that the dose found in the
plant matter thereafter is sufficient for transmission by the natural oral route.
Dead carcasses of positive animals or body parts thereof can be sources of infection.
CWD is not highly infectious. Sometimes natural exposure to the agent for 90 days straight is
barely enough to transmit disease. Mathiason et al. 2009 PLoS ONE
4(6)e5916,doi:10.1371/journal.pone.005916
Most TSE computer models have failed the test of accuracy over time. The early predictions
from the BSE models in Europe predicted 54,000 human deaths from variant CJD. The total
stands at less than 150 several decades later.( Each life lost was missed by someone and was a
tragedy).
The early computer models for CWD predicted that it would decimate the nation’s entire herd.
Computer models were very efficient in genning up fear, public outcry, and research funding.
Cases of CWD are terminal when followed to the end of the course of the disease. Most CWD
positive deer die before ever exhibiting clinical symptoms.
Certain genotypes coding for long incubation periods or resistance to infection may be being
selected for in the wild. This may in time strengthen the nation’s deer herd.
CWD does not affect other livestock by natural transmission methods. No evidence exists to
refute this statement.
There is NO evidence that eating venison from positive deer is a public health hazard. A case
history of a large number of individuals who were accidentally fed venison from a positive deer
is recorded from the Verona Fire Department in Oneida, NY. Those individuals have all been
followed for ten years and the study terminated this year with no incidence of zoonotic disease.
Man has likely been eating scrapie positive meat from sheep or goats for over 200 years.
Australia is free of scrapie and the incidence of human TSEs is no different there than in
countries that have scrapie.
There is NO evidence that CWD or any other TSE can be transmitted by semen or embryos.
There are case studies and very efficient models of CWD management and control strategies that
have been used by other states, e.g. PA,NY.
The pathology of CWD and BSE in the brains of infected subjects is distinct from each other.
The incidence of CWD in free range cervids is higher than the incidence in captive cervids by a
fourfold factor based on USDA statistics.
CWD respects no borders. CWD positive mule deer walked across the TX-NM border several
years ago.
There is no difference in harvest rates between infected and non infected individuals. Grear et al.
2006 quoted by Kroll, James C. White paper CWD : the issues at hand. July 2013
CWD has a higher incidence in males than in females.
TSEs as disease agents are RARE.
Rabies in deer is very rare. CWD is more rare than cervid rabies.
Examples, by contrast, of highly infectious diseases of deer that are very virulent and cause
significant mortality would include epizootic hemorrhagic disease( EHD), a viral agent, and in
south Texas, Anthrax, a bacterial infection caused by Bacillus anthracis. Anthrax is also a potent
public health risk.
The rate of infection or at least the progression of the disease is slowed in animals that have
unique amino acid sequences at various loci in their genetic code. Some species of cervids are
more susceptible than others. The mule deer appears up to ten times more susceptible than the
whitetail.
In other species TSEs are documented that are spontaneous or sporadic in nature, i.e. there is no
casual epidemiology. The normal proteins that are present in healthy bodies apparently change
and begin to misfold. In fact with the most well studied human TSE, Creutzfeldt-Jakob Disease
(CJD), approximately 80 % of the cases are sporadic/spontaneous. The incidence of this disease
is rare also. Approximately one case per million persons is well documented. Other TSEs are
familial in man. In CJD, genotype plays a critical role in susceptibility.
Task three:
How do we balance the interests of the deer breeding industry with preventing the spread of the
disease?
The deer breeding industry is ranked among the top ten agricultural commodities in the state of
Texas. Recent surveys place its value at over at over $1 billion per annum and it is an integral
part of the multibillion dollar hunting industry.
There are currently several models that have been used effectively for control.There is a national
model. Based on known incubation periods, a five year trace back and trace forward period
seems to be reasonable.
The comments herein are based on science, known elements of the disease, coupled with
experience.
As I understand the numbers:
A 2.0+ year old buck was the index case. He was raised on the index ranch. There were 126 deer
that entered the index facility. 26 remain there.
Priority one:
Freeze movement of any connected ranch inbound or outbound for now.Allow unrelated ranches
in TX to sign affidavits attesting to unrelatedness and apply for “movement qualified”.
Geography and proximity have less value than relatedness. Ranchers who are unrelated and who
only release stockers to their own high fence property that is contiguous to their pens are of
lowest priority.
Test antemortem and post mortem all of the inbound group or account for them. This can be
easily done by transfer permit computer search. Likely some of the 100 that came in and left
have been tested prior to June 30,2015.
Test all remaining deer at index facility.
Analyze results. If all results are “not detected/not positive” we may be documenting a
spontaneous case of CWD or one transmitted by carrion eaters, predators, or via contaminated
plant matter shipped in and used as forage.
If the index ranch suffers any positives then downstream ranches move into heightened priority
status and are dealt with in the same manner as the index ranch.
If results are “not detected/not positive” on the index ranch, the downstream ranches are now the
remaining concern .
In the meantime, any downstream ranch that voluntarily sacrifices those related animals in toto
via official means and tissue collections and whose test results are returned as “not detected/not
positive” should be able to apply for “ movement qualified”. The balance of downstream
individuals should be livability verified or confirmed dead and tested by rectal biopsy and codon
testing at position 95/96 if deemed an additional risk by authorities.
If any downstream animal turns up positive then that facility now becomes activated for
additional investigation using the same logic as above.
Respectfully submitted.


Are idiots multiplying faster than normal people?[Linked Image]
Re: CWD in Texas [Re: Frio County Hunts] #5835356 07/17/15 01:55 AM
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CWD.....Decision up same post over & over

Last edited by SheepHunter; 07/17/15 02:37 AM.

Lucky 7 Exotic Ranch located in Eden, Tx. Well managed self sustaining herds roaming our 3,000 acre ranch. First Class Lodging, Ranch style meals and qualified guides. 30+ species.
Re: CWD in Texas [Re: Frio County Hunts] #5835363 07/17/15 01:58 AM
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That's good stuff right there. No evidence of humans being infected from consuming meat from an infected deer. I think the state did a stand up job at looking at all the facts.

Re: CWD in Texas [Re: Frio County Hunts] #5835686 07/17/15 12:31 PM
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Thanks stxranchman. Nice to see a post from someone with facts and common sense solutions. This makes me proud to be a Texan to see we don't follow, we lead. cheers texas


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Re: CWD in Texas [Re: Frio County Hunts] #5835690 07/17/15 12:34 PM
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Amen!

Re: CWD in Texas [Re: Frio County Hunts] #5835839 07/17/15 02:17 PM
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Friday, July 17, 2015

TPW Commission Holds Special Meeting on Chronic Wasting Disease

http://chronic-wasting-disease.blogspot.com/2015/07/tpw-commission-holds-special-meeting-on.html


kind regards, terry

Re: CWD in Texas [Re: Frio County Hunts] #5835857 07/17/15 02:29 PM
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You really shouldn't attach your blather to official links. It's bad form IMO.

Really? "Texas fell to mad cow disease"??

I guess I slept thru that.


Crotchety old bastidge
Re: CWD in Texas [Re: therancher] #5835872 07/17/15 02:37 PM
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kind of a big deal
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Originally Posted By: therancher
You really shouldn't attach your blather to official links. It's bad form IMO.

Really? "Texas fell to mad cow disease"??

I guess I slept thru that.


A UK immigrant died of vCJD earlier this year. He relocated to TX but still traveled a lot to the UK and Middle East


Bottom line, never trust a man whose uncle was eaten by cannibals.-Sen Joni Ernst
Re: CWD in Texas [Re: BOBO the Clown] #5836145 07/17/15 05:24 PM
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Originally Posted By: BOBO the Clown
Originally Posted By: therancher
You really shouldn't attach your blather to official links. It's bad form IMO.

Really? "Texas fell to mad cow disease"??

I guess I slept thru that.


A UK immigrant died of vCJD earlier this year. He relocated to TX but still traveled a lot to the UK and Middle East


Yeah, and I'm fairly certain you and I agree that "Texas fell to mad cow disease" is probably a tad extreme in that description.

And imbedding his blather in technical linked information is IMO poor form.

Last edited by therancher; 07/17/15 05:30 PM.

Crotchety old bastidge
Re: CWD in Texas [Re: Frio County Hunts] #5836156 07/17/15 05:32 PM
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And then some...

Not saying I would cry over another mad cow scare. Made a lot of money last time it temporarily depressed the market


Bottom line, never trust a man whose uncle was eaten by cannibals.-Sen Joni Ernst
Re: CWD in Texas [Re: LuckyHunter] #5836181 07/17/15 05:46 PM
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Originally Posted By: SheepHunter
CWD.....Decision up same post over & over


clap funny clip


Marc C. Helfrich
Retirement Planner

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Re: CWD in Texas [Re: BOBO the Clown] #5837667 07/18/15 10:40 PM
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Originally Posted By: BOBO the Clown
Originally Posted By: therancher
You really shouldn't attach your blather to official links. It's bad form IMO.

Really? "Texas fell to mad cow disease"??

I guess I slept thru that.


A UK immigrant died of vCJD earlier this year. He relocated to TX but still traveled a lot to the UK and Middle East



Sunday, November 23, 2014 Confirmed Variant Creutzfeldt-Jakob Disease (variant CJD) Case in Texas in June 2014 confirmed as USA case NOT European

Confirmed Variant Creutzfeldt-Jakob Disease (variant CJD) Case in Texas

Updated: October 7, 2014

CDC and the Texas Department of State Health Services (DSHS) have completed the investigation of the recently reported fourth vCJD case in the United States. It confirmed that the case was in a US citizen born outside the Americas and indicated that the patient's exposure to the BSE/vCJD agent most likely occurred before he moved to the United States; the patient had resided in Kuwait, Russia and Lebanon. The completed investigation did not support the patient's having had extended travel to European countries, including the United Kingdom, or travel to Saudi Arabia. The specific overseas country where this patient’s infection occurred is less clear largely because the investigation did not definitely link him to a country where other known vCJD cases likely had been infected.

http://www.cdc.gov/ncidod/dvrd/vcjd/other/confirmed-case-in-texas.htm

>>>the patient had resided in Kuwait, Russia and Lebanon.

>>>The completed investigation did not support the patient's having had extended travel to European countries, including the United Kingdom, or travel to Saudi Arabia.

NOW we all know why the state of Texas or the CDC did not want to report this case, because it was a home grown case of nvCJD right here in Texas?...tss

Monday, June 02, 2014

Confirmed Variant CJD Case in Texas

http://vcjd.blogspot.com/2014/06/confirmed-variant-cjd-case-in-texas.html Saturday, July 18, 2015

SPONTANEOUS TRANSMISSIBLE SPONGIFORM ENCEPHALOPATHY TSE PRION AKA MAD COW TYPE DISEASE, DOES IT EXIST NATURALLY IN THE FIELD?

http://transmissiblespongiformencephalop...spongiform.html

Re: CWD in Texas [Re: therancher] #5837684 07/18/15 10:57 PM
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Originally Posted By: therancher
You really shouldn't attach your blather to official links. It's bad form IMO.

Really? "Texas fell to mad cow disease"??

I guess I slept thru that.



I guess you did sleep through the Texas mad cow case that was finally confirmed after the great state of Texas tried covering it up. took 7 months and an act of congress to get that mad cow case finally confirmed. see for yourself ;


1st mad cow that got away $$$

FOR IMMEDIATE RELEASE Statement May 4, 2004 Media Inquiries: 301-827-6242 Consumer Inquiries: 888-INFO-FDA

Statement on Texas Cow With Central Nervous System Symptoms

On Friday, April 30 th , the Food and Drug Administration learned that a cow with central nervous system symptoms had been killed and shipped to a processor for rendering into animal protein for use in animal feed.

FDA, which is responsible for the safety of animal feed, immediately began an investigation. On Friday and throughout the weekend, FDA investigators inspected the slaughterhouse, the rendering facility, the farm where the animal came from, and the processor that initially received the cow from the slaughterhouse.

FDA's investigation showed that the animal in question had already been rendered into "meat and bone meal" (a type of protein animal feed). Over the weekend FDA was able to track down all the implicated material. That material is being held by the firm, which is cooperating fully with FDA.

http://www.fda.gov/bbs/topics/news/2004/NEW01061.html


ONE HUNDRED EIGHTH CONGRESS

COMMITTEE ON GOVERNMENT REFORM

May 13,2004

The Honorable Ann M. Veneman Secretary of Agriculture Department of Agriculture1400 Independence Avenue, SW Washington, DC 20250

Dear Madam Secretary:

I am writing to express concern that the recent failure of the U.S. Department of Agriculture (USDA) to test a Texas cow with neurological syrnptoms for bovine spongifonnencephalopathy (BSE) may reflect wider problems in the surveillance program. USDA apparently does not keep track of how many cows condemned for central nervous system symptoms are tested for BSE nor does it require that suspect carcasses be held pending testing...

FULL TEXT ;

http://oversight.house.gov/documents/20040607142914-86912.pdf


The 2nd mad cow that almost got away, brain samples laid up somewhere for 7 months, so the BSE MRR risk policy, so the BSE MRR risk policy, the legal trading of all strains of TSE prions was ratified. after an act of Congress and scientist from around the world complaining about it to the OIG, the sample was finally sent to Weybridge and CONFIRMED. ...


Release No. 0336.05 Contact: USDA Jim Rogers 202-690-4755 FDA Rae Jones 301-827- 6242

Printable version Email this page

U.S. Department of Agriculture (USDA) Food and Drug Administration (FDA)

Investigation Results of Texas Cow That Tested Positive for Bovine Spongiform Encephalopathy (BSE) Aug. 30, 2005

The U.S. Department of Agriculture's Animal and Plant Health Inspection Service (APHIS) and the U.S. Department of Health and Human Services' Food and Drug Administration (FDA) have completed their investigations regarding a cow that tested positive for bovine spongiform encephalopathy (BSE) in June 2005. The agencies conducted these investigations in collaboration with the Texas Animal Health Commission and the Texas Feed and Fertilizer Control Service.

Our results indicate that the positive animal, called the index animal, was born and raised on a ranch (termed the "index farm") in Texas. It was a cream colored Brahma cross approximately 12 years old at the time of its death. It was born prior to the implementation of the 1997 feed ban instituted by FDA to help minimize the risk that a cow might consume feed contaminated with the agent thought to cause BSE. The animal was sold through a livestock sale in November of 2004 and transported to a packing plant. The animal was dead upon arrival at the packing plant and was then shipped to a pet food plant where it was sampled for BSE. The plant did not use the animal in its product, and the carcass was destroyed in November 2004.

APHIS attempted to trace all adult animals that left the index farm after 1990, as well as all progeny born within 2 years of the index animal's death. Together, these animals are called animals of interest.

During the course of the investigation, USDA removed and tested a total of 67 animals of interest from the farm where the index animal's herd originated. All of these animals tested negative for BSE. 200 adult animals of interest were determined to have left the index farm. Of these 200, APHIS officials determined that 143 had gone to slaughter, two were found alive (one was determined not to be of interest because of its age and the other tested negative), 34 are presumed dead, one is known dead and 20 have been classified as untraceable. In addition to the adult animals, APHIS was looking for two calves born to the index animal. Due to record keeping and identification issues, APHIS had to trace 213 calves. Of these 213 calves, 208 entered feeding and slaughter channels, four are presumed to have entered feeding and slaughter channels and one calf was untraceable.

To determine whether contaminated feed could have played a role in the index animal's infection, FDA and the Texas Feed and Fertilizer Control Service conducted a feed investigation with two main objectives: 1) to identify all protein sources in the animal=s feed history that could potentially have been the source of the BSE agent, and 2) to verify that cattle leaving the herd after 1997 were identified by USDA as animals of interest and were rendered in compliance with the 1997 BSE/ruminant feed rule.

The feed history investigation identified 21 feeds or feed supplements that were used on the farm since 1990. These feed ingredients were purchased from three retail feed stores and were manufactured at nine feed mills. This investigation found that no feed or feed supplements used on the farm since 1997 were formulated to contain prohibited mammalian protein. Due to this finding, FDA has concluded that the animal was most likely infected prior to the 1997 BSE/ruminant feed rule.

The investigation into the disposition of herd mates from this farm involved visits to nine slaughter plants and eight rendering plants. The investigation found that all of the rendering plants were operating in compliance with the BSE/ruminant feed rule. A review of the inspection history of each of these rendering firms found no violations of the FDA feed ban rule.

APHIS and FDA are very pleased with the results of their investigations, which show the animals of interest did not present a threat to livestock and that the ruminant feed rule is being followed. The U.S. maintains an interlocking system of safeguards designed to prevent BSE from entering the human and animal food chain. USDA also remains vigilant in its attempt to find BSE in the United States. To date, there have been more than 450,000 animals tested in the last 14 months and only two BSE positive animals found in this country.

For more information on USDA's epidemiological investigation and a copy of the report, please visit the APHIS website at http://www.aphis.usda.gov/lpa/issues/bse/bse.html or

http://www.aphis.usda.gov/lpa/issues/bse/epi-updates/bse_final_epidemiology_report.pdf

For more information on FDA's feed investigation, please visit the FDA's website at

http://www.fda.gov/cvm/texasfeedrpt.htm

Last Modified: 08/31/2005

http://www.usda.gov/wps/portal/usdahome?contentidonly=true&contentid=2005/08/0336.xml

Aug 30, 2005 USDA Texas BSE Investigation—Final Epidemiology Report

http://www.aphis.usda.gov/newsroom/hot_issues/bse/downloads/bse_final_epi_report8-05.pdf


TSS REPORT ON 2ND TEJAS MAD COW Mon, 22 Nov 2004 17:12:15 -0600 (the one that did NOT get away, thanks to the Honorable Phyllis Fong)


-------- Original Message --------

Subject: Re: BSE 'INCONCLUSIVE' COW from TEXAS ???

Date: Mon, 22 Nov 2004 17:12:15 –0600

From: "Terry S. Singeltary Sr." To: Carla Everett References: <[log in to unmask]> <[log in to unmask] us>

Greetings Carla,still hear a rumor;

Texas single beef cow not born in Canada no beef entered the food chain?

and i see the TEXAS department of animal health is ramping up forsomething, but they forgot a url for update?I HAVE NO ACTUAL CONFIRMATION YET...can you confirm???terry

==============================

-------- Original Message --------

Subject: Re: BSE 'INCONCLUSIVE' COW from TEXAS ???

Date: Fri, 19 Nov 2004 11:38:21 –0600

From: Carla Everett To: "Terry S. Singeltary Sr." References: <[log in to unmask]>

The USDA has made a statement, and we are referring all callers to the USDA web site. We have no information about the animal being in Texas. Carla

At 09:44 AM 11/19/2004, you wrote:

>Greetings Carla,

>>i am getting unsubstantiated claims of this BSE 'inconclusive' cow is from

>TEXAS. can you comment on this either way please?

>>thank you,

>Terry S. Singeltary Sr.

>>

===================

-------- Original Message --------

Subject: Re: BSE 'INCONCLUSIVE' COW from TEXAS ???

Date: Mon, 22 Nov 2004 18:33:20 -0600 From: Carla Everett

To: "Terry S. Singeltary Sr." References: <[log in to unmask]> <[log in to unmask] us> <[log in to unmask]> <[log in to unmask] us> <[log in to unmask]>

our computer department was working on a place holder we could postUSDA's announcement of any results. There are no results to be announced tonightby NVSL, so we are back in a waiting mode and will post the USDA announcement when we hear something.

At 06:05 PM 11/22/2004, you wrote:>why was the announcement on your TAHC site removed?

>>Bovine Spongiform Encephalopathy:

>November 22: Press Release title here

>>star image More BSE information

>>>>terry

>>Carla Everett wrote:

>>>no confirmation on the U.S.' inconclusive test...

>>no confirmation on location of animal.

>>>>>>

==========================

THEN, 7+ MONTHS OF COVER-UP BY JOHANN ET AL! no doubt about it now $$$

NO, it's not pretty, hell, im not pretty, but these are the facts, take em or leave em, however, you cannot change them.

with kindest regards,

I am still sincerely disgusted and tired in sunny Bacliff, Texas USA 77518

Terry S. Singeltary Sr.

FULL 130 LASHINGS TO USDA BY OIG again

http://www.usda.gov/oig/webdocs/50601-10-KC.pdf

Link: TSS

http://lists.ifas.ufl.edu/cgi-bin/wa.exe?A2=ind0612&L=sanet-mg&T=0&P=23557


News Release Texas Animal Health Commission Box l2966 * Austin, Texas 78711 * (800) 550-8242 * FAX (512) 719-0719 Bob Hillman, DVM * Executive Director For info, contact Carla Everett, information officer, at 1-800-550-8242, ext. 710, or ceverett@tahc.state.tx.us

For immediate release---

State-Federal Team Responds to Texas BSE Case

The US Department of Agriculture announced June 29 that genetic testing has verified that an aged cow that tested positive for Bovine Spongiform Encephalopathy or BSE originated from a Texas beef cattle herd. Tissues for laboratory testing were initially collected from the animal in November 2004, and the carcass was incinerated and did not enter the human food, animal feed or fertilizer supply system. While tests in November indicated the animal did not have BSE, retesting in England in June confirmed the animal had the disease. The Texas Animal Health Commission (TAHC), the state’s livestock and poultry health regulatory agency, and USDA have jointly assigned a state-federal team to conduct the epidemiological investigation and response.

“The TAHC and US Department of Agriculture’s Veterinary Services are working with a complement of experts from federal and state animal health, food safety, public health and feed regulatory agencies to ensure the continued safety and wholesomeness of our meat supply,” said Dr. Bob Hillman, Texas state veterinarian and executive director of the TAHC, the state’s livestock and poultry health regulatory agency. “Epidemiological investigations are thorough and focus on verifying the herd of origin, and when, where and how the animal and potentially, any herd mates, were exposed to the abnormal prion, or disease agent, that causes BSE. Additionally, epidemiology investigations trace the infected animal’s movement and herd mates. Animals potentially exposed to the disease will be depopulated, with proper disposal. The animals will not be introduced into the human or animal food chain.”

The USDA’s BSE testing protocol requires testing of emaciated or injured cattle, cattle that exhibit central nervous system disorder, cattle unable to rise or to walk normally, and cattle that die of unknown causes. Since June 1, 2004, brain tissue samples from more than 394,000 cattle have been tested in the U.S. and were negative for BSE. Of those, 38,320 were tested in Texas, Dr. Hillman noted. BSE surveillance has been conducted in the U.S. since l990.

The U.S. has taken preventive measures against the introduction of BSE since l989, when prohibitions were placed on cattle and other ruminants from BSE-affected countries, noted Dr. Hillman. In 1997, the importation ban was extended to all of Europe.

Dr. Hillman said the U.S. Food and Drug Administration (FDA) in 1997 banned the use of ruminant-derived protein (from animals such as cattle and sheep) in feed for cattle and other ruminants. There is no evidence that BSE spreads from live animal to animal in the herd, but cattle can be exposed by eating feed that contains rendered protein from infected animals. “These measures taken by the USDA and the FDA are safeguards that work to protect livestock, and ultimately, our meat supply,” he said.

--30--

http://www.tahc.state.tx.us/news/pr/2005/2005Jun30_BSE_Positive_Results.pdf

THE USDA JUNE 2004 ENHANCED BSE SURVEILLANCE PROGRAM WAS TERRIBLY FLAWED ;

CDC DR. PAUL BROWN TSE EXPERT COMMENTS 2006

In an article today for United Press International, science reporter Steve Mitchell writes:

Analysis: What that mad cow means

By STEVE MITCHELL UPI Senior Medical Correspondent

WASHINGTON, March 15 (UPI) -- The U.S. Department of Agriculture was quick to assure the public earlier this week that the third case of mad cow disease did not pose a risk to them, but what federal officials have not acknowledged is that this latest case indicates the deadly disease has been circulating in U.S. herds for at least a decade.

The second case, which was detected last year in a Texas cow and which USDA officials were reluctant to verify, was approximately 12 years old.

These two cases (the latest was detected in an Alabama cow) present a picture of the disease having been here for 10 years or so, since it is thought that cows usually contract the disease from contaminated feed they consume as calves. The concern is that humans can contract a fatal, incurable, brain-wasting illness from consuming beef products contaminated with the mad cow pathogen.

"The fact the Texas cow showed up fairly clearly implied the existence of other undetected cases," Dr. Paul Brown, former medical director of the National Institutes of Health's Laboratory for Central Nervous System Studies and an expert on mad cow-like diseases, told United Press International. "The question was, 'How many?' and we still can't answer that."

Brown, who is preparing a scientific paper based on the latest two mad cow cases to estimate the maximum number of infected cows that occurred in the United States, said he has "absolutely no confidence in USDA tests before one year ago" because of the agency's reluctance to retest the Texas cow that initially tested positive.

USDA officials finally retested the cow and confirmed it was infected seven months later, but only at the insistence of the agency's inspector general.

"Everything they did on the Texas cow makes everything they did before 2005 suspect," Brown said.

Despite this, Brown said the U.S. prevalence of mad cow, formally known as bovine spongiform encephalopathy, or BSE, did not significantly threaten human or cattle health.

"Overall, my view is BSE is highly unlikely to pose any important risk either in cattle feed or human feed," he said.

However, Jean Halloran of Consumers Union in Yonkers, N.Y., said consumers should be troubled by the USDA's secrecy and its apparent plan to dramatically cut back the number of mad cow tests it conducts.

"Consumers should be very concerned about how little we know about the USDA's surveillance program and the failure of the USDA to reveal really important details," Halloran told UPI. "Consumers have to be really concerned if they're going to cut back the program," she added.

Last year the USDA tested more than 300,000 animals for the disease, but it has proposed, even in light of a third case, scaling back the program to 40,000 tests annually.

"They seem to be, in terms of actions and policies, taking a lot more seriously the concerns of the cattle industry than the concerns of consumers," Halloran said. "It's really hard to know what it takes to get this administration to take action to protect the public."

The USDA has insisted that the safeguards of a ban on incorporating cow tissue into cattle feed (which is thought to spread the disease) and removal of the most infectious parts of cows, such as the brain and spinal cord, protect consumers. But the agency glosses over the fact that both of these systems have been revealed to be inadequately implemented.

The feed ban, which is enforced by the Food and Drug Administration, has been criticized by the Government Accountability Office in two reports, the most recent coming just last year. The GAO said the FDA's enforcement of the ban continues to have weaknesses that "undermine the nation's firewall against BSE."

USDA documents released last year showed more than 1,000 violations of the regulations requiring the removal of brains and spinal cords in at least 35 states, Puerto Rico and the Virgin Islands, with some plants being cited repeatedly for infractions. In addition, a violation of similar regulations that apply to beef exported to Japan is the reason why Japan closed its borders to U.S. beef in January six weeks after reopening them.

Other experts also question the adequacy of the USDA's surveillance system. The USDA insists the prevalence of mad cow disease is low, but the agency has provided few details of its surveillance program, making it difficult for outside experts to know if the agency's monitoring plan is sufficient.

"It's impossible to judge the adequacy of the surveillance system without having a breakdown of the tested population by age and risk status," Elizabeth Mumford, a veterinarian and BSE expert at Safe Food Solutions in Bern, Switzerland, a company that provides advice on reducing mad cow risk to industry and governments, told UPI.

"Everybody would be happier and more confident and in a sense it might be able to go away a little bit for (the USDA) if they would just publish a breakdown on the tests," Mumford added.

UPI requested detailed records about animals tested under the USDA's surveillance plan via the Freedom of Information Act in May 2004 but nearly two years later has not received any corresponding documents from the agency, despite a federal law requiring agencies to comply within 30 days. This leaves open the question of whether the USDA is withholding the information, does not have the information or is so haphazardly organized that it cannot locate it.

Mumford said the prevalence of the disease in U.S. herds is probably quite low, but there have probably been other cases that have so far gone undetected. "They're only finding a very small fraction of that low prevalence," she said.

Mumford expressed surprise at the lack of concern about the deadly disease from American consumers. "I would expect the U.S. public to be more concerned," she said.

Markus Moser, a molecular biologist and chief executive officer of Prionics, a Swiss firm that manufactures BSE test kits, told UPI one concern is that if people are infected, the mad cow pathogen could become "humanized" or more easily transmitted from person to person.

"Transmission would be much easier, through all kinds of medical procedures" and even through the blood supply, Moser said.

© Copyright 2006 United Press International, Inc. All Rights Reserved

http://www.upi.com/ConsumerHealthDaily/view.php?StoryID=20060315-055557-1284r

http://www.upi.com/Science_News/2003/12/...47861072816318/

CDC - Bovine Spongiform Encephalopathy and Variant Creutzfeldt ... Dr. Paul Brown is Senior Research Scientist in the Laboratory of Central Nervous System ... Address for correspondence: Paul Brown, Building 36, Room 4A-05, ...

http://www.cdc.gov/ncidod/eid/vol7no1/brown.htm

PAUL BROWN COMMENT TO ME ON THIS ISSUE

Tuesday, September 12, 2006 11:10 AM

"Actually, Terry, I have been critical of the USDA handling of the mad cow issue for some years, and with Linda Detwiler and others sent lengthy detailed critiques and recommendations to both the USDA and the Canadian Food Agency." ........TSS

http://madcowtesting.blogspot.com/2009/07/mad-cow-cover-up-usa-masked-as-sporadic.html

OR, what the Honorable Phyllis Fong of the OIG found ;

Audit Report Animal and Plant Health Inspection Service Bovine Spongiform Encephalopathy (BSE) Surveillance Program ­ Phase II and Food Safety and Inspection Service

Controls Over BSE Sampling, Specified Risk Materials, and Advanced Meat Recovery Products - Phase III

Report No. 50601-10-KC January 2006

Finding 2 Inherent Challenges in Identifying and Testing High-Risk Cattle Still Remain

http://www.usda.gov/oig/webdocs/50601-10-KC.pdf

Subject: USDA OIG SEMIANNUAL REPORT TO CONGRESS FY 2007 1st Half (bogus BSE sampling FROM HEALTHY USDA CATTLE)

Date: June 21, 2007 at 2:49 pm PST

Owner and Corporation Plead Guilty to Defrauding Bovine Spongiform Encephalopathy (BSE) Surveillance Program

An Arizona meat processing company and its owner pled guilty in February 2007 to charges of theft of Government funds, mail fraud, and wire fraud. The owner and his company defrauded the BSE Surveillance Program when they falsified BSE Surveillance Data Collection Forms and then submitted payment requests to USDA for the services. In addition to the targeted sample population (those cattle that were more than 30 months old or had other risk factors for BSE), the owner submitted to USDA, or caused to be submitted, BSE obex (brain stem) samples from healthy USDA-inspected cattle. As a result, the owner fraudulently received approximately $390,000. Sentencing is scheduled for May 2007.

snip...

Topics that will be covered in ongoing or planned reviews under Goal 1 include:

soundness of BSE maintenance sampling (APHIS),

implementation of Performance-Based Inspection System enhancements for specified risk material (SRM) violations and improved inspection controls over SRMs (FSIS and APHIS),

snip...

The findings and recommendations from these efforts will be covered in future semiannual reports as the relevant audits and investigations are completed.

4 USDA OIG SEMIANNUAL REPORT TO CONGRESS FY 2007 1st Half

http://www.usda.gov/oig/webdocs/sarc070619.pdf

THIS is just ONE month report, of TWO recalls of prohibited banned MBM, which is illegal, mixed with 85% blood meal, which is still legal, but yet we know the TSE/BSE agent will transmit blood. we have this l-BSE in North America that is much more virulent and there is much concern with blood issue and l-BSE as there is with nvCJD in humans. some are even starting to be concerned with sporadic CJD and blood, and there are studies showing transmission there as well. ... this is one month recall page, where 10 MILLION POUNDS OF BANNED MAD COW FEED WENT OUT INTO COMMERCE, TO BE FED OUT. very little of the product that reaches commerce is ever returned via recall, very, very little. this was 2007, TEN YEARS AFTER THE AUGUST 4, 1997, PARTIAL AND VOLUNTARY MAD COW FEED BAN IN THE USA, that was nothing but ink on paper. i have listed the tonnage of mad cow feed that was in ALABAMA in one of the links too, this is where the infamous g-h-BSEalabama case was, a genetic relation matching the new sporadic CJD in the USA. seems this saga just keeps getting better and better.......$$$

10,000,000+ LBS. of PROHIBITED BANNED MAD COW FEED I.E. BLOOD LACED MBM IN COMMERCE USA 2007

snip...see full text ;

Tuesday, November 02, 2010

IN CONFIDENCE

The information contained herein should not be disseminated further except on the basis of "NEED TO KNOW".

BSE - ATYPICAL LESION DISTRIBUTION (RBSE 92-21367) statutory (obex only) diagnostic criteria CVL 1992

http://bse-atypical.blogspot.com/2010/11/bse-atypical-lesion-distribution-rbse.html

2009 UPDATE ON ALABAMA AND TEXAS MAD COWS 2005 and 2006

http://bse-atypical.blogspot.com/2006/08/bse-atypical-texas-and-alabama-update.html


I'm just saying...

Re: CWD in Texas [Re: Frio County Hunts] #5837692 07/18/15 11:03 PM
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whew!.. I need a burger after that long reply! grin

Re: CWD in Texas [Re: Frio County Hunts] #5837895 07/19/15 02:18 AM
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Flounder, no one here believes Texas fell to mad cow. Stop posting non sense links. Take off your tin foil hat and relax.

That being said, ranch in medina tried getting the tpwd to sign an agreement on the amount of deer they would kill. Tpwd declined, shocking, so basically the situation is right where it started.

Re: CWD in Texas [Re: flounder] #5838022 07/19/15 04:43 AM
Joined: Jul 2013
Posts: 7,192
T
therancher Offline
THF Trophy Hunter
Offline
THF Trophy Hunter
T
Joined: Jul 2013
Posts: 7,192
Flounder, you do realize that more people die of drowning in 5 gallon buckets in Texas each year than die of CJD right?

So, I guess "Texas 'fell' to 5 gallon buckets" too?

Quitit!


Crotchety old bastidge
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