Endemic chronic wasting disease causes mule deer population decline in Wyoming

Endemic chronic wasting disease causes mule deer population decline in Wyoming

''Our findings support CWD as a population-limiting disease of mule deer with the potential to cause dramatic declines that resemble local population extinction.''

''Other studies have found a negative association between CWD prevalence and λ [11,12,40,42], but none have documented λ estimates resulting from endemic CWD as low as those reported here.''

http://journals.plos.org/plosone/article?id=10.1371/journal.pone.0186512


SATURDAY, OCTOBER 21, 2017

Endemic chronic wasting disease causes mule deer population decline in Wyoming

http://chronic-wasting-disease.blogspot.com/2017/10/endemic-chronic-wasting-disease-causes.html


with sad regards, terry

How can any one put their name on a population study that’s has a span of less then 10 years.

Sad.

Don't believe everything you read. We hunt the endemic area and deer numbers are high with great buck/doe ratios and even better fawn /doe ratios. Been seeing more doe fawns groups this year than before and later season mean big bucks will be taken. Stay home and leave them to our friends if you're worried.
That study was on private land that is heavily outfitted, old news not supported by local G&F biologists and scientists.

What time period would be adequate ? I have no idea is why I ask.

As long as this particular sky has been falling, you would think it would have hit by now.

ALot more then 10 years, 50 would be a better number. 100 would put you back to almost extinct mule deer numbers though. Wyoming averages a bad winter kill atleast once every 10 years and also bad drought atleast once every ten years. Kind of hard to show population tends in such a minute time frame

Flounder is a vegetarian... per him all ungulats have some form of CWD, mad cow, scrappies and it all turns into vCJD

there ya go again BOBO the Clown, talking about something you know nothing about or you just like to make stuff up, take your pick. please show us where i have stated i am a vegetarian, as you stated? you can't, i have never said it, what i have stated, i don't want to eat tse prions, and you should not either, they can kill you and yours. please stop making stuff up or lying BOBO the clown, thank you...kind regards, terry

You have hammered for mass execution of entire unglulates populations as best standard. Not test, not monitoring... entire elimination of whole populations.

You pushed government denial and cover up, so it make sense if you choose not to eat unglulates. Well you many not be a vegetarian chickens and fish don’t have any know pinions. Yet

there ya go again BOBo the clown, always talking about something you know nothing about. it's PRIONS Sir, prions. you might want to study up a bit before commenting. i refuse to go through the menu of what i eat here, especially for someone that is so clueless.

BOBo the clown, you do realize how much slaughterhouse waste and hospital waste is deposited in our waterways? NEVER MIND, i forgot i was speaking with BOBo the clown, a by-product of the industry, clueless BOBo the clown. don't count your chickens or your fishes before the protein has flipped Sir. ...terry


Wednesday, July 10, 2013

Prions in the Ocean: A Natural Case of Prion Disease in Dolphins

http://transmissiblespongiformencephalop...e-of-prion.html

SE1806

TRANSMISSION STUDIES OF BSE TO DOMESTIC FOWL BY ORAL EXPOSURE TO BRAIN HOMOGENATE

1 challenged [censored] bird was necropsied (41 months p.i.) following a period of ataxia, tremor, limb abduction and other neurological signs. Histopathological examination failed to reveal any significant lesions of the central or peripheral nervous systems...

snip...

94/01.19/7.1


http://www.bse.org.uk/files/yb/1994/01/19007001.pdf

http://webarchive.nationalarchives.gov.uk/20060525120000/http://www.bseinquiry.gov.uk/evidence/yb/1994jan.htm

also,

TRANSLATION

F437/91

A CONTRIBUTION TO THE NEUROPATHOLOGY OF THE RED-NECKED OSTRICH (STRUTHIO CAMELUS) - SPONGIFORM ENCEPHALOPATHY -

* The Red-Neck Ostrich 'THE AUTOPSY' & TSEs

THE AUTOPSY

Date: Mon, 11 Jun 2001 16:24:51 -0700
Reply-To: Bovine Spongiform Encephalopathy
Sender: Bovine Spongiform Encephalopathy
From: "Terry S. Singeltary Sr." Subject: The Red-Neck Ostrich & TSEs 'THE AUTOPSY'

######## BSE #########

TRANSLATION

F437/91

A CONTRIBUTION TO THE NEUROPATHOLOGY OF THE RED-NECKED OSTRICH (STRUTHIO CAMELUS) - SPONGIFORM ENCEPHALOPATHY -

H A Schoon, Doris Brunckhorst and J Pohienz Institute of Pathology, Veterinary University of Hannover

Introduction

Since the first appearance of BSE in Great Britain in l985 {review in TRUYEN & KAADEN, l990), research into the incidence, diagnosis, differential diagnosis and epidemiology of spongiform encephalopathies in humans and animals has been a focus of medical and public interest. In view of the growing number of reports of "new" spontaneously or experimentally susceptible species (cats: WYATT et al, l990; pigs: DAWSON et al, 1990), and of the associated questions with regard to the causal agent and in particular its transmissibility, it seems essential that agnopathogenetic individual cases should also be described. We therefore report below the preliminary findings of morphological examinations of three red-necked ostriches in 1986, 1988 and 1989, taking account of differential diagnostic factors.

History/subjects

The three ostriches (Flock A: Ostrich 1, female, adult, 150 kg; Flock B: Ostrich 2, female, adult, 80 kg; Ostrich 3: male, juvenile, 60 kg) came from two zoos in North West Germany and were euthenised because of their hopeless prognosis. Preliminary reports indicated that all three birds had presented protracted central nervous symptoms with ataxia, disturbance of balance and discoordinated feeding behaviour. Ostrich 2 had also exhibited pronounced lameness of the left lower limbs and the juvenile bird was suffering from perosis. The birds were fed on vegetable material, supplemented by commercial compound poultry feed and ''raw meat'', some of which was ''obtained from local small emergency slaughterers''. Comparable clinical pictures with fatal outcome in individual birds had occurred in both flocks: in a male bird at the same time (Flock A) and in several ostriches over recent years (Flock B).

Methods

Autopsy was followed in all three cases by histopathological examination of the following tissues: heart (several locations including coronary arteries and aorta), right and left pulmonary lobes, liver, kidneys, limb musculature, peripheral nerves (brachial plexus, sciatic nerve, in each case both left and right) and brain (left and right cerebral hemispheres, two samples each from the cranial/caudal third, two sagittal sections of the cerebellum, two cross-sections of the brain stem at the level of the optical lobes, four cross-sections from the medulla oblongata). The tissue material was fixed in formalin and embedded in Paraplast by the conventional method and the sections were evaluated using the following staining techniques and histochemical reactions: all organs: haematoxylin eosin staining; brain: PAS reaction (McManus), Ziehl/Neelsen staining (mod. Pearse), iron method (Lillie) for detection of neuromelanin, Turnbull's reaction (Bancroft & Stevens), alkaline Congo red method (Puchtler) (of SCOON & SCHINKEL, 1986), myelin sheath staining (Spielmeyer) (ROMEIS, 1968). In addition, unstained sections were examined by fluorescence microscopy (to detect autofluorescing lipofuscin granula) and the following lipid stains were applied to cryostat sections of liver, and of heart and skeletal musculature: Sudan III, Sudan black, oil red.

Findings


snip...


A CONTRIBUTION TO THE NEUROPATHOLOGY OF THE RED-NECKED OSTRICH (STRUTHIO CAMELUS) - SPONGIFORM ENCEPHALOPATHY


http://www.bseinquiry.gov.uk/files/sc/Seac10/tab06.pdf

https://web.archive.org/web/20060210125137/http://www.bseinquiry.gov.uk/files/sc/Seac10/tab06.pdf

OPINION on : NECROPHAGOUS BIRDS AS POSSIBLE TRANSMITTERS OF TSE/BSE ADOPTED BY THE SCIENTIFIC STEERING COMMITTEE AT ITS MEETING OF 7-8 NOVEMBER 2002

OPINION

1. Necrophagous birds as possible transmitters of BSE. The SSC considers that the evaluation of necrophagous birds as possible transmitters of BSE, should theoretically be approached from a broader perspective of mammals and birds which prey on, or are carrion eaters (scavengers) of mammalian species. Thus, carnivorous and omnivorous mammals, birds of prey (vultures, falcons, eagles, hawks etc.), carrion eating birds (crows, magpies etc.) in general could be considered possible vectors of transmission and/or spread of TSE infectivity in the environment. In view also of the occurrence of Chronic Wasting Disease (CWD) in various deer species it should not be accepted that domestic cattle and sheep are necessarily the only source of TSE agent exposure for carnivorous species. While some information is available on the susceptibility of wild/exotic/zoo animals to natural or experimental infection with certain TSE agents, nothing is known of the possibility of occurrence of TSE in wild animal populations, other than among the species of deer affected by CWD in the USA.

1 The carrion birds are animals whose diet regularly or occasionally includes the consumption of carcasses, including possibly TSE infected ruminant carcasses.

C:\WINNT\Profiles\bredagi.000\Desktop\Necrophagous_OPINION_0209_FINAL.doc


http://ec.europa.eu/food/fs/sc/ssc/out295_en.pdf


terry

This guy seems like a raving lunatic.

Well there you go, you just made your very own case as to why you should be vegan.

Congrats on the new safe life style

Don't catch this.

Kuru. This disease is seen in New Guinea. It's caused by eating human brain tissue contaminated with infectious prions. Because of increased awareness about the disease and how it is transmitted, kuru is now rare.

Study by Canadian Food Inspection Agency shows transmission of CWD by eating infected whitetail deer meat into 3 of 5 macaque monkeys.

Y’all can ignore/poo-poo it if you like. I don’t.

It also stated in the reporting that we consume 7-15k CWD infected a deer a year. So NA should be having 4200-9000 new human deaths a year solely to vCJD/CWD.

If you are stressing that much you should have ALLl animals consumed tested.

Don't Eat human brain tissue from New Guinea!

Is it ok to eat it from anywhere other than New Guinea?

I don't know but you'll catch Kuru in New Guinea. I'll stick to barbacoa from the local taqueria.

.

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